#39 Excess unbound iron is very bad! Get PROPERLY tested

by Morley Robbins on July 20, 2016

MAG-pie & MAG-net Alert!


Please share this “oldie, but goodie” with your favorite M.ineral D.enialist:


And I quote from another article that I was just reading, Griffith et al, 2009, “Acquired Copper Deficiency: A Potentially Serious & Preventable Complication Following Gastric Bypass Surgery”:

“Mitochondrial cytochrome c oxidase, which plays a critical role in the transfer of Iron to the cytosol for incorporation into Heme [23]. Ceruloplasmin (i.e., ferroxidase enzyme) contributes to anemia as it is ESSENTIAL [emphasis added] in the loading of transferrin with Iron in the Liver and Ceruloplasmin in markedly diminished in parallel with Copper [24].” (Please note, the linked article is Citation [23] noted above…)

It is WELL KNOWN in Iron research circles that LOW Hemoglobin is a clinical sign of Copper deficiency (i.e. LOW BIOAVAILABLE COPPER) . And I will NOW go out on a limb and assert that LOW Ferritin, LOW serum Iron and LOW % Saturation are indicative of Copper deficiency, AS WELL. What is clearly emerging from the literature is that these 3 markers noted ^^^^ are HIGHLY CORRELATED and HIGHLY INDICATIVE of what is called “Anemia of Chronic Illness,” or “Anemia of Inflammation.”

They are NOT indications of “Iron deficiency…”

They ARE Signs of “Iron dysregulation…” that is fueled by LOW bioavailable Copper as BEST expressed by LOW Ceruloplasmin.

Please do your level best to explain that PHYSIOLOGICAL TRUTH to your doctor. This I(ron)gnorance must STOP.

And for those that want to see it in “red,” please order this test:

Just know, you can NOT properly interpret a set of Iron markers without knowing Mag, RBC, Plasma Zinc, Serum Copper & Serum Ceruloplasmin. It is ESSENTIAL to have an understanding of the minerals that REGULATE Iron, in addition to Iron itself…

OK, back to our “regular” programming…

A votre sante!

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